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It was noted that any dietary recommendations for the use of PUFA or MUFA in preference to the other should be based on outcomes other than cholesterol concentrations. Carbohydrate foods differ substantially in their quantitative and qualitative features with regard to micronutrients, phytochemicals, fibers, and other bioactive substances, which could in turn have differential effects on plasma lipids and CHD risk.
SFA intake was lower in the intervention group vs.Nulla varius consequat magna, id molestie ipsum volutpat quis. Fusce id mi diam, non ornare orci.
Among healthy subjects, replacement of saturated fat with carbohydrates resulted in reductions of TC and LDL-C across sex and ethnicity 114. Triglyceride concentrations tended to be lower with the MUFA diet, but were significantly higher with the high carbohydrate diet.
Of note, the dietary lipid responses varied on the basis of baseline lipid profiles, but they did not differ by metabolic syndrome or insulin resistance status 103.
Mixed results have been reported in diabetic patients with respect to the effects of dietary carbohydrates on plasma lipids 115.
A recent systematic analysis of 17 clinical trials among obese subjects reported that low-carbohydrate diets were associated with significant decreases in TG and increases in HDL-C, without impacting LDL-C 121.
Studies replacing saturated fat with carbohydrates have investigated the impact of carbohydrate intake on LDL particle size, which has been associated with CVD endpoints 125-129.
Individuals with pattern B have a higher proportion of small dense LDL particles, and thus more likely to have atherogenic dyslipidemia.
The amount of dietary carbohydrates was associated with a decrease in LDL particle size and an increase in LDL density, contributing to the atherogenic dyslipidemia 132. Of note, proportions of saturated vs.
One third of these individuals converted to pattern B on this diet.